Cardiac fibrosis, a pathological phenomenon, manifests as an abnormal buildup of collagen fibers within the heart tissue, resulting in heightened stiffness and diminished cardiac function. This condition is often a consequence of prolonged cardiovascular diseases, including conditions like hypertension or chronic inflammation. The replacement of normal heart tissue with collagen deposits compromises the heart's efficiency in pumping blood, ultimately elevating the risk of heart failure. In the realm of cardiology, cardiac fibrosis stands as a substantial concern, serving as a pivotal factor in the progression of various heart disorders. To address this challenge, a comprehensive understanding of the molecular mechanisms triggering cardiac fibrosis is imperative. This knowledge is instrumental in the development of precise therapeutic interventions designed to prevent or reverse the detrimental effects of cardiac fibrosis, thereby safeguarding overall heart function. Current research endeavors in this domain aim to unravel the intricate complexities associated with cardiac fibrosis, fostering the discovery of innovative treatments that can effectively mitigate its impact on cardiovascular health.